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Our product range contains a wide range of Montelukast Sodium, Alendronate Sodium, VALSARTAN SODIUM, Rabeprazole Sodium and Losartan Potassium
Prevention And Long-term Treatment Of Asthma . It Is Also Used In Certain Patients To Relieve Allergy Symptoms (Eg, Itchy, Runny, Or Stuffy Nose; Sneezing) And To Prevent Asthma Attacks Caused By Exercise. It Is A Leukotriene Receptor Antagonist. It Works By Blocking A Substance Called Leukotriene, Which Helps To Decrease Certain Asthma And Allergy Symptoms. Montelukast Is A Leukotriene Receptor Antagonist (Ltra) Used For The Maintenance Treatment Of Asthma And To Relieve Symptoms Of Seasonal Allergies. This Reduces Bronchoconstriction Otherwise Caused By The Leukotriene And Results In Less Inflammation. It Is Usually Administered Orally. Montelukast Blocks The Action Of Leukotriene D4 On The Cysteinyl Leukotriene Receptor Cyslt1 In The Lungs And Bronchial Tubes By Binding To It. This Reduces The Broncho- Constriction Otherwise Caused By The Leukotriene And Results In Less Inflammation. Again Because Of Its Very Specific Locus Of Operation, It Does Not Interact With Other Allergy Medications Such As Theophylline.
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Alendronate Sodium is a Bisphosphonate that Acts as a Specific Inhibitor of Osteoclast-mediated Bone Resorption. Bisphosphonates are Synthetic Analogs of Pyrophosphate that Bind to the Hydroxyapatite Found in Bone. Alendronate Sodium is Chemically Described as (4-amino-1-hydroxy Butylidene) Bisphosphonic Acid Monosodium Salt Trihydrate. A Drug Used to Treat Certain Bone Conditions, such as Osteoporosis and Paget Disease of the Bone. It is also Being Studied in the Treatment of Hypercalcemia (high Levels of Calcium in the Blood) and Bone Pain Caused By Cancer. Alendronate Sodium Slows the Breakdown of Bone and Prevents the Loss of Calcium. It is a Type of Bisphosphonate.treatment of Osteoporosis in Postmenopausal Women; Prevention of Osteoporosis in Postmenopausal Women At Risk of Developing Osteoporosis; Increase Bone Mass in Men; Treatment of Glucocorticoid-induced Osteoporosis in Men and Women, Treatment of Paget Disease of the Bone. and It is Prescribed for Both Men and Women Who have Developed a Form of Osteoporosis Sometimes Caused By Steroid Medications such as Prednisone.
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Valsartan Is An Orally Active Nonpeptide Triazole-derived Antagonist Of Angiotensin (At) Ii With Antihypertensive Properties. Valsartan Selectively And Competitively Blocks The Binding Of Angiotensin Ii To The At1 Subtype Receptor In Vascular Smooth Muscle And The Adrenal Gland, Preventing At Ii-mediated Vasoconstriction, Aldosterone Synthesis And Secretion And Renal Reabsorption Of Sodium And Resulting In Vasodilation, Increased Excretion Of Sodium And Water, A Reduction In Plasma Volume And A Reduction In Blood Pressure. There Is Contradictory Evidence With Regard To Treating People With Heart Failure With A Combination Of An Angiotensin Receptor Blocker Like Valsartan And An Angiotensin-converting Enzyme Inhibitor, With Two Major Clinical Trials (Charm-additive & Valheft) Showing A Reduction In Death, And Two Others (Valiant And Ontarget) Showing No Benefits, And More Adverse Effects Including Heart Attacks.
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Rabeprazole Sodium Is Substituted Benzimidazole Proton Pump Inhibitor, With Potential Anti-ulcer Activity. After Protonation, Accumulation, And Transformation To The Active Sulfenamide Within The Acidic Environment Of Gastric Parietal Cells, Rabeprazole Selectively And Irreversibly Binds To And Inhibits The H+, K+atpase (Hydrogen-potassium Adenosine Triphosphatase) Enzyme System Located On The Parietal Cell Secretory Surface, Inhibiting Gastric Acid Secretion. Short-term Treatment In Healing And Symptomatic Relief Of Duodenal Ulcers And Erosive Or Gastroesophageal Reflux Disease (Gerd); Maintaining Healing & Reducing Relapse Rates Of Heartburn Symptoms In Patients(Gerd), Long-term Treatment Of Pathological Hypersecretory Conditions, Including Zollinger-ellison Syndrome To Eradicate Helicobacter Pylori. Gastric Ulcer (Gu), Peptic Ulcer Disease (Pud) Maintenance Of Healing Of Erosive Or Ulcerative Gerd Healing.
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Losartan Potassium Is The Potassium Salt Of Losartan, Belongs To Non- Peptide Group- Angiotensin Ii Receptor Antagonist With Antihypertensive Activity. Losartan Selectively And Competitively Binds To The Angiotensin Ii Receptor (Type At1) And Blocks The Binding Of Angiotensin Ii To The Receptor, Thus Promoting Vasodilatation And Counteracting The Effects Of Aldosterone. Converted From Angiotensin I By Angiotensin-converting Enzyme (Ace), Angiotensin Ii Stimulates The Adrenal Cortex To Synthesize And Secrete Aldosterone, Decreasing Sodium Excretion And Increasing Potassium Excretion, And Acts As A Vasoconstrictor In Vascular Smooth Muscle. These Type Of Medications Work By Preventing The Constriction (Narrowing) Of Blood Vessels (Veins And Arteries). Losartan Was The First Angiotensin Ii Receptor Antagonist To Be Marketed. It Keeps Blood Vessels From Narrowing, Which Lowers Blood Pressure And Improves Blood Flow. Losartan Is Used To Treat High Blood Pressure (Hypertension). It Is Also Used To Lower The Risk Of Stroke In Certain People With Heart Disease. Losartan Is Used To Slow Long Term Kidney Damage In People With Type 2 Diabetes Who Also Have High Blood Pressure.
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Risedronate Sodium Is The Sodium Salt Of Risedronic Acid, A Synthetic Pyridinyl Bisphosphonate. Risedronic Acid Binds To Hydroxyapatite Crystals In Bone And Inhibits Osteoclast-dependent Bone Resorption. Biphosphonate, A Salt, Ester, Or Anion Of A Dimer Of Phosphonic Acid (Diphosphonic Acid), Inhibits Bone Resorption As A Sodium Salt And Complexed With Technetium Tc 99m For Bone Imaging. Monophosphonates Are Not Active. Biphosphonates Are Used In Disorders Affecting The Skeleton Such As Osteoporosis, Metastatic Disease And Paget's Disease. While Bisphosphonates Are Analogues Of Endogenous Pyrophosphate Structurally, They Are Characterized By A P-c-p Bond, The Linking Oxygen Is Replaced With A Carbon, Which Is Resistant To Enzymatic And Chemical Hydrolysis. It Is Used For The Treatment Of Postmenopausal Osteoporosis In Women By Building Healthy Bone And Restoring Some Of The Bone Loss As A Result Of Osteoporosis Include Vitamin D And Calcium Supplements.
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Treating Osteoporosis (weak Bones) in Women Who are Postmenopause. Ibandronate is a Bisphosphonate and Works By Slowing Bone Loss and Allowing New Bone to Be Formed. Ibandronic Acid or ibandronate Sodium is a Potent Bisphosphonate Drug Used in the Prevention and Treatment Of osteoporosis and Metastasis-associated Skeletal Fractures in Multiple Myeloma, Breast Cancer. it May also Be Used to Treat hypercalcemia-elevated Blood Calcium levels side Chains Creates Chemical Physicla Properties, Mechanism of Resorption, and Pharmacokinetics. Biphosphonates Bind Strongly to Hydroxyapatite Crystals At the Sites of Increased Bone Turnover Preferentially and Inhibit the Formation, Aggregation, and Dissolution of the Crystals.they also May Inhibit Osteoclast Function Directly, Promotion of Osteoclast Apoptosis, and Interference with Osteoblast- Mediated Osteoclast Activation.
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Zoledronic acid anhydrous is a bisphosphonate. The chemical classifi- cation of zoledronic acid anhydrous is diphosphonates anhydrous form of a synthetic imidazole third generation bisphosphonate analog of pyrophosphate with antiresorptive activity. Zoledronate binds to hydroxyapatite crystals in the bone matrix & inhibits farnesyl pyro- phosphate (diphosphate) synthase, thereby preventing protein preny- lation within the mevalonate pathway. This leads to the loss of downs-tream metabolites essential for osteoclast function, leading to the induction of apoptosis and eventually, osteoclast-cell death. By pre- venting osteoclast-mediated bone resorption zoledronate decreases bone turnover and stabilizes the bone matrix.
