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    Tripcit-BRD Tablets

    • Type Of MedicineAllopathic
    • Cool & Dry PlaceAs Prescribed By The Doctor
    • FeaturesNo Side Effect, Quick Action
    • StorageCool & Dry Place
    • Supply TypeSupplier, Retailer
    • Preferred Buyer Location All over the world
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    • calendar Member Since 7 Years
    • building Nature of Business Retailer
    • gst icon GST No. 04AANFC1119P1ZC

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    • Salt NameTrypsin, Bromelain, Rutoside Diclofenac Sodium
    • SolubilityHigh
    • Type Of MedicineAllopathic
    • FeaturesNo Side Effect, Quick Action
    • StorageCool & Dry Place
    • Cool & Dry PlaceAs Prescribed By The Doctor

    Class : Trypsin and Bromelain belongs to the class of naturally occuring systemic enzymes and Rutoside Trihydrate is a phytonutrient. Diclofenac belongs to the class of non-steroidal anti-inflammatory agent (NSAID) with antipyretic and analgesic actions.
    Indication
    OBETRA-D Tablets can be used in the treatment of following conditions :

    • Post-operative pain.
    • Edema and inflammation in contusions, lacerations, cuts, sinusitis, otitis, externa, PID (Pelvic inflammatory disease), tooth extraction, cellulitis, abscess, sport injuries, sprains and surgery.
    • Painful osteoarthritis, rheumatoid arthritis, spondylopathies, tendonitis, polymyositis, dermatomyositis and gout.
    • Pain management of kidney stones and gallstones, chronic pain in cancer, post-traumatic pain, dysmenorrhea and acute migraines.
    • Temporary relief from aches, pains and muscle soreness due to everyday activity.


    Dosage : One OBETRA-D Tablet three times a day or as directed by Physician.
    Mechanism of action : Trypsin and Bromelain are proteolytic enzymes. They break peptide bond inside protein molecules. Bromelain exerts anti-inflammatory activity by affecting prostaglandin synthesis. Rutoside is a glycoside and has antioxidant effect. It increases the strength of the walls of the blood capillaries and regulates their permeability. Diclofenac inhibit both leukocyte migration and the enzyme cylooxygenase (COX-1 and COX-2), leading to the peripheral inhibition of prostaglandin synthesis.


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